IL1B and atherosclerosis: And the research of Qiao et al. (48) have also revealed that the new mechanism of inflammatory body NLRP3 is regulated in macrophages and atherosclerosis, so as to provide new insights into the role of autophagy-lysosome pathway in atherosclerosis, indicating that the pharmacological activation of CMA may provide new treatment strategies for the atherosclerosis and other inflammatory body NLRP3/IL-1β driven diseases.