Moreover, diBSP01 exhibited almost no antitumor activity with no significant difference compared with the vehicle control in the transiently STING knocked out zebrafish while diBSP01 still showed a significant tumor inhibitory effect in normal wild-type zebrafish (data not shown), which implied that diBSP01 exerted antitumor efficacy in zebrafish in a STING-dependent manner and suggested that diBSP01 could activate the zebrafish STING protein. The gene discussed is STING1; the disease is neoplasm.