Indeed, mitochondria are central players in the loss of β‐cell function related to type 2 diabetes (Las et al., 2020); in addition to their role regulating intracellular ATP production in response to glucose levels (and consequent insulin secretion), changes in mitochondrial genetics, bioenergetics, redox state, structure, and dynamics are involved in β‐cell functional regulation (Las et al., 2020). This evidence concerns the gene INS and type 2 diabetes mellitus.