Besides, exosomes of AMSCs infected by miR-199a lentivirus have successfully delivered miR-199a to hepatocellular carcinoma (HCC) cells, and remarkably improved sensitivity of chemotherapeutic agents to cancer cells for the improvement of HCC chemosensitivity via the inhibition of the mammalian target of rapamycin (mTOR) signaling pathway [158]. This evidence concerns the gene MTOR and hepatocellular carcinoma.