Human PCD patients with a mutation of CCDC151, CCDC114, or TTC25 lose DNAH5 (human OAD γ-HC) from the entire length of respiratory cilia (Hjeij et al., 2014; Onoufriadis et al., 2013; Wallmeier et al., 2016). This evidence concerns the gene ODAD1 and primary ciliary dyskinesia.