IL17A and Stroke: Recent immunological research has indicated that inflammatory mechanisms are central to the pathophysiology of stroke.12 In particular, the highly conserved pro-inflammatory cytokine Interleukin-17A (IL-17A) holds key functions in the initiation of the excessive detrimental inflammatory response in the ischaemic brain in the first hours and days.13,14 In stroke, γδ T cells are the main source of IL-17A.