The neutrophil antimicrobial peptide LL37 and dsDNA in the serum of SLE patients are able to form immune complexes (ICs) as autoantigens, which can in turn activate B cells through B-cell receptors and TLR9 in order to produce anti-DNA and anti-LL37 antibodies [143]. The gene discussed is CAMP; the disease is systemic lupus erythematosus.