CEBPB and Parkinson disease: In accordance with our study showing a transient activation of Cebpb during NFs-mediated rescue from apoptosis [43], previous studies demonstrated that up-regulation of this TF in rat primary cortical and cerebellar neuronal cultures plays neuroprotective and antiapoptotic effects, while its reduced neuronal levels may represent a pathogenic factor in neurodegenerative disorders, including Alzheimer’s and Parkinson’s diseases, supporting the potential of Cebpb as a pharmacological target in brain injury and neurodegenerative disorders (Figure 1 and Figure 3) [141,147,148,149,150,151,152].