These disorders include Alzheimer’s disease (tau and amyloid), Parkinson’s disease (-synuclein), Huntington’s disease (huntingtin), frontotemporal dementia (FTLD-Tau), and prion diseases (prion protein), whose molecular signatures were found to be increasing in B. schlosseri in late-cycle before takeover (present data) [7]. This evidence concerns the gene HTT and juvenile Huntington disease.