TGFB1 and polyostotic fibrous dysplasia: Despite the lack of PFD literary data on experimental models of allergic asthma, two previous studies [33,34] suggested a curative potential for PFD in the classic asthma phenotype by suppressing the BALF’s inflammatory cytokine and platelet-derived growth factor, inhibiting TGF-β1 expression, and reducing the goblet cell number & subepithelial fibrosis.