The majority of the stomach adenocarcinoma samples, 64% and 71%, were diploid for NAGS and CPS1 genes, respectively; 20% and 16% of the stomach adenocarcinoma samples exhibited gains of NAGS and CPS1, respectively; 13% and 10% of stomach adenocarcinoma samples exhibited shallow deletions of NAGS and CPS1, respectively; and the remaining stomach adenocarcinoma samples had either deep deletions or amplifications of the two genes (Figure 4M). Here, NAGS is linked to gastric adenocarcinoma.