Meanwhile, tumors escaping from NK cell-mediated immunosurveillance may also be achieved by inducing the expression of inhibitory receptors such as NKG2A on NK cells [39], as well as promoting the transdifferentiation from NK cells to other intermediate ILC1 subsets with reduced anti-tumor function in the presence of elevated levels of transforming growth factor-β (TGF-β) [40]. Here, TGFB1 is linked to neoplasm.