In addition, the proliferation and growth of tumor cells are supported by mTOR-enhancing glucose metabolism by upregulating the transporter 1 (GlUT1), HIF1-α, and c-MYC, resulting in the enhancement of glycolytic enzymes, such as enolase (ENO), phosphofructokinase (PFK), and phosphoglucoisomerase (PGI) [72,73,74]. Here, MTOR is linked to neoplasm.