GIST has been shown to overexpress a wide variety of receptors, such as an abundance of somatostatin 2A (SST2A) receptors in Cajal neuronal cells [124], as well as an upregulation of neurotensin 1 receptors, that are present regardless of the GIST degree of mutation [125]. This evidence concerns the gene NTSR1 and gastrointestinal stromal tumor.