The results of this study showed that diabetic patients with combined hyperlipidaemia had a lower control rate compared to diabetic patients without any complications (37.24% vs. 47.67%) because free fatty acids, as precursors to hepatic glucose metabolism, increase apoptosis of pancreatic β-cells and increase muscle insulin resistance[28], leading to impaired insulin secretion and persistent hyperglycaemia [29], which requires diabetic patients to be aware of abnormalities in their lipid metabolism while controlling their blood glucose. The gene discussed is INS; the disease is Hyperglycemia.