It was ultimately identified that the majority of patients with this presentation had high-risk APOL1 genotypes, thus predisposing them to glomerular injury in the setting of high inflammatory stress often seen with viral illnesses in general, not necessarily due to direct viral invasion.18,19 It remains to be proven, then, that CKD will be a long-term sequelae following COVID-19 independent of AKI, which was not the primary finding of the present analysis. This evidence concerns the gene APOL1 and chronic kidney disease.