In addition, immune subtypes inflammatory (C3), IFN-λ(C2),wound healing (C1), immunologically quiet (C5), lymphocyte depleted(C4), and TGF-β dominant (C6) analysis revealed an associationbetween SLC6A20, particularly with inflammatory (C3)and IFN-λ (C2) signatures in THYM, READ, COAD, ESCA, and OVsamples, suggesting a potential involvement of SCL6A20 in immune modulation in these cancer types. This evidence concerns the gene SLC6A20 and cancer.