NFKB1 and autoimmune thrombocytopenic purpura: A recent study found that complement c4d/c5b deposition on BMMSCs from ITP patients was associated with increased apoptosis, possibly due to complement-derived IL-1β/IL-1R/NF-κB, ERK1/2 and p38 MAPK signaling pathways [25], which has previously shown to be involved in MSC proliferation and migration differentiation [26].