The total number of cellular interactions in patients with AIDP decreased compared to HC (Supplementary Fig. S5a,b; Fig. 3c), but the number of cellular interactions between several cell types (CD16 + monocytes and CD8 + T, CD16 + monocytes, NK, IgA plasma B, platelets, etc.)significantly increased (Fig. 3a,c). This evidence concerns the gene FCGR3A and Guillain-Barre syndrome, familial.