AKT1 and idiopathic pulmonary fibrosis: Accordingly, the relevant pathways, such as protein digestion and absorption, ECM‐receptor interaction, focal adhesion, PI3K‐Akt signaling, protein processing in endoplasmic reticulum, complement and coagulation cascades, and cytokine–cytokine receptor interaction were upregulated in the lungs of IPF patients (Figure 6C).