HOTAIR and gastric cancer: In GC, lncRNA HOTAIR is overexpressed and promotes GC by acting as a ceRNA by sponging miR-331-3p [41], lncRNA EIF3J-DT enhances GC chemoresistance by autophagy activation [42], and hypoxia-induced lncRNA-CBSLR influences the progress of GC via regulating ferroptosis [43].