By contrast, for long-term transient interference with CaMKII signaling, our results further support the previously described memory erasure (14): Although our expression analysis indicated significantly less expression of the T286D/T305/306AA mutant compared to the K42M tool mutant or to wildtype, the in vivo expression of all of these CaMKII forms was completely extinguished on day 9 after infection with the corresponding HSV-based expression vectors. This evidence concerns the gene CAMK2G and infection.