In addition, a recent paper found that m6A reader HNRNPC can regulate Treg cell abundance as a possible mechanism for m6A methylation-mediated response against CTLA-4, indicating that activation of the immune microenvironment by targeting m6A regulators may serve as a potential therapeutic approach for advanced PCa(52).Our study synthetically analyzed the relationship between the expression of m6A regulators and immune characteristics and drug sensitivity of PCa patients. The gene discussed is CTLA4; the disease is posterior cortical atrophy.