In addition, it was shown that suppression of BCL2L14 protected kidney epithelial cells from detrimental effects of glucose and oxygen deprivation, and limited nephrotoxicity of cisplatin [89], attenuated UV-induced apoptosis in prostate carcinoma and breast cancer cells [83, 84], and prevented apoptosis induced by ectopic FAU overexpression in human T-lymphoblastic leukemia cells and embryonic kidney-derived cells [54]. Here, BCL2L14 is linked to prostate carcinoma.