Although more than 80% of patients experience mild symptoms, a substantial proportion can be critically ill, especially those who are older and those with comorbidities.1,2,3 The pathogenesis of COVID-19 involves a dysregulated host immune response and excessive inflammation, which can lead to acute respiratory distress syndrome and multiorgan failure.4,5,6 A subgroup of patients with severe COVID-19 show hyperinflammatory features, with increased circulating levels of cytokines, including interleukin (IL)–1 and IL-6,3,5 which are associated with poor outcomes.7,8. Here, IL6 is linked to COVID-19.