PTH and systemic lupus erythematosus: By further establishing the SLE-GIOP mouse model, it is speculated that the mechanism of JP in the prevention and treatment of SLE-GIOP may be related to the protection of the hypothalamus–pituitary–adrenal axis from the inhibition of exogenous steroid hormones, the promotion of endogenous F secretion, the inhibition of PTH secretion or activity, the promotion of intestinal calcium absorption, and reduction of urinary calcium excretion [5].