In particular, alterations in the function of the vestibular nerveand/or the labyrinth could be due to changes in vascular permeability,26 toxin release by the VS,27 changes in axonalmyelination,28 and cellularinflammation mediated by the tumour’s micro-environment.29 Future work could examine the relationship betweenclinical vestibular dysfunction in patients with NF2-SWN and these putative mechanism bymeasuring tumour and serum biomarkers30 in addition to metrics that quantify vestibular function anddisability. This evidence concerns the gene NF2 and neoplasm.