Another scRNA-seq analysis of murine cardiac ECs after MI showed that the EC clusters upregulated genes related to cardiac muscle morphogenesis (Myl2, Myl3, Mb, Tnnt2, Actc1, and Tnni3), indicating that cardiac ECs may switch on cardiomyogenic genes in response to MI (Li et al., 2019). This evidence concerns the gene TNNT2 and myocardial infarction.