Notably, the inflammatory status was indistinguishable between EAE WT and EAE miR-142-HE conditions (Cd3e, Gfap, Aif1, Tnf: EAE-WT vs. EAE-HE, p >0.05; Fig. 2A), although miR-142-HE mice were protected from the EAE-induced synaptic damage. The gene discussed is GFAP; the disease is hereditary elliptocytosis.