TNF and hereditary elliptocytosis: Consistently with what was observed in HE mice, TNF was unable to induce significant increases of glutamatergic activity in anti-miR condition, whereas it promoted the expected excitotoxic synaptopathy in the scramble condition (decay time and half width: SCRM-TNF vs. anti-miR-TNF: p <0.0001; Fig. 4A).