BCL2 and metabolic dysfunction-associated steatotic liver disease: The acridone derivative A22 (7,Figure 2) (Li et al., 2020) stimulated Bcl-2 expression, reducing hepatocyte apoptosis and alleviating inflammation, endoplasmic reticulum stress, and cirrhosis in a NAFLD/NASH (non-alcoholic fatty liver disease/non-alcoholic steatohepatitis) model.