CRP and atrial fibrillation: In addition, C-reactive protein, interleukin-6, tumor necrosis factor-α, and other inflammatory factors can cause an abnormal electrical activity of pulmonary veins, shorten atrial action potential, and interact with heat shock protein or myeloperoxidase to promote atrial fibrosis [6], thereby promoting the occurrence and recurrence of atrial fibrillation and thromboembolic events.