As can be noticed from the results, AL137789.1 silencing elevated the protein expression of E-cadherin yet diminished those of N-cadherin and Vimentin in PCa cells BxPC3 and PANC1 (Figure 5a–h, p < 0.001), while overexpression of AL137789.1 promoted the protein expressions of N-cadherin and Vimentin but repressed that of E-cadherin (Figure 5a–h, p < 0.001). This evidence concerns the gene VIM and posterior cortical atrophy.