Subdivision of the CD64+ population demonstrated that G2 monocytes accounted for ~80% of all iNOS-expressing cells in both WT and ΔRD1 infection (Fig. 7E and F), indicating that Ly6C+MHCII+ monocytes are the main producers of iNOS-derived nitric oxide in the infected tissue. Here, FCGR1A is linked to infection.