On the other hand, hs-cTnT has been evaluated in patients seeking care for chest pain in absence of myocardial infarction, showing that high hs-cTnT values can mirror myocardial injury in other types of myocardial injuries than MI, reflecting the complex biology of the cardiac sarcomeres of the cardiac myocytes [5–8]. This evidence concerns the gene TNNT2 and myocardial infarction.