For this, we took advantage of the fact that vRNP levels increase during infection (Kawakami et al., 2011), and analysed viral inclusions over a time course, in two conditions: with endogenous levels of Rab11a (using cells expressing GFP, as in Alenquer et al., 2019), and overexpressing Rab11a (in the form of GFP-Rab11a, as in Alenquer et al., 2019; Figure 3A–B, Figure 3—figure supplement 2). This evidence concerns the gene RAB11A and infection.