We noted that the neutrophils from tumors generated in qMCP−/−; Ntv-a mice appeared to be enriched in interactions with many tumor clusters (T0, and T2 to T5) through secretion of tumor necrosis factor α (TNF-α) and signaling via TNF-α receptor-I (TNFR-I: p55) and DAG1 on tumor cells (Fig. 5D). This evidence concerns the gene DAG1 and neoplasm.