To the contrary, COVID-19 binds the ACE2 receptor and prevents Ang1-7 production from AngII, thereby creating an imbalance in the ratio of AngII to Ang1-7 that is associated with the COVID-19-asscociated pro-inflammatory response within the lungs (Zbinden-Foncea et al., 2020; De Sousa et al., 2021). This evidence concerns the gene AGT and COVID-19.