Concretely, CXCL11 stimulation upregulated circUBAP2 expression in tumor cells, and the later counteracted miR-4756-mediated inhibition on interferon-induced protein with tetratricopeptide repeats (IFIT)1/3 by sponging miR-4756, resulting in upregulation of IFIT1/3 expression that contributed to IL-17 and IL-1β expression, and elevated the migration capability of HCC cells. The gene discussed is IFIT1; the disease is neoplasm.