Administration of H2O2-preconditioned UC-MSCs reduced lung remodeling by attenuating expression of α-smooth muscle actin (α-SMA) and TGF-β as well as myeloperoxidase (MPO) activity in experimental bleomycin-induced lung fibrosis (Mahmoudi et al., 2020). This evidence concerns the gene TGFB1 and pulmonary fibrosis.