Indeed, in mice with a CA2/CA3 region specific OXTR knock-down, the neonatal administration of OXT did not rescue social alterations (Pan et al., 2022) while, in Dysbindin-KO mice, a mouse model of schizophrenia with altered OXTR levels in the CeA, the manipulation of OXTR within this region rescued emotional recognition deficits (Ferretti et al., 2019). The gene discussed is DTNBP1; the disease is schizophrenia.