In human NASH specimens, ATF4 and SLC7A11 are also correlated (Fig. 7I), supporting our finding that inhibition of ferroptosis by the ATF4–SLC7A1 axis suppresses hepatocarcinogenesis in NASH-afflicted MUP-uPA mice (Fig. S7F). This evidence concerns the gene SLC7A1 and metabolic dysfunction-associated steatohepatitis.