More specifically, MC SULT1E1+ exhibited strong cell‐cell communication with other MC subpopulations in many cancer‐associated signaling pathways, including the non‐canonical WNT and NCAM pathways (Figure 3E), suggesting that MC SULT1E1+ functions as a core modulator of tumor behavior by regulating other tumor cells. The gene discussed is NCAM1; the disease is neoplasm.