This might also explain higher associations between the DSMQ-R scales and HbA1c in T1D [consistent with previous findings (23)], where glycemic outcomes directly depend on the consistent coordination and adjustment of carbohydrate intake, activities and insulin doses; whereas in T2D, glycemic control may rely more on diet and activity and less on glucose checking and meal-specific decisions (depending on the treatment regimen); also, residual insulin action may stabilize glycemic levels and reduce hyperglycemia. The gene discussed is INS; the disease is Hyperglycemia.