In support of that hypothesis, we show (1) suppression of PP1A or PKC isoforms partially regulates MerlinS13 phosphorylation (Fig. 3i, j), (2) epistatic MerlinS13D or MerlinΔNTD rescue partially restores Wnt signaling in meningioma cells (Fig. 3c, e), and (3) the non-canonical Wnt pathway regulators AMOT, AMOTL1, AMOTL2, DSG2, and DLG1 were identified in proximity to Merlin alongside β-catenin in meningioma cells38-40 (Supplementary Table 2). The gene discussed is AMOT; the disease is meningioma.