S100A8 and infection: Supplementary Figure S4B shows that the H. pylori infection of AGS cells did not significantly kill the gastric cells during the time of the assay, as we observed less than 3% of 7-AAD positive cells. This result also reduces the possibility that the chemoattraction could be explained by the non-specific release of the damage-associated molecular pattern (DAMP) molecules. Altogether, these data support that an active infection of gastric epithelial cells by virulent H. pylori CagA(+) strains would attract EBV-infected B lymphocytes to the site of infection.