We could show that these rhCCK ligands, for example [nat/177Lu]Lu-(R)-DOTAGA-rhCCK-16, revealed up to 8-fold increased activity levels in the tumor but also approximately 30-fold higher levels in the kidney when compared to [nat/177Lu]Lu-DOTA-PP-F11N, despite its distinctly lower CCK-2R affinity [19]. Here, CCKBR is linked to neoplasm.