Since stabilization of c-MYC by different USPs in different cancers is underpinned by direct physical interaction between both molecules [22,23,27] including USP37 [27], it is, therefore, also conceivable that in the ABC subtype of DLBCL, c-MYC is likely regulated by direct interaction with USP37. The gene discussed is MYC; the disease is diffuse large B-cell lymphoma.