For instance, genetic factors involved in the pathogenesis of GBS could be human leukocyte antigen (HLA) genes, cluster of differentiation (CD) 1A, FAS, Fc gamma receptors (FcGR), intercellular adhesion molecule-1 (ICAM1), different interleukins, nucleotide oligomerization domain (NOD), toll-like receptor 4 (TLR4), or tumor necrosis factor-α (TNF-α) [16]. This evidence concerns the gene FAS and Guillain-Barre syndrome.