In glioblastoma, miRNA-155 regulates AGTR1 expression after transcription and blocks the AGTR1/NF-κB/CXCR4 signaling axis, resulting in a decrease in the levels of AGTR1 and CXCR4 and attenuating AGTR1-mediated angiogenesis, EMT, stem cell signal transduction, and MAPK signal transduction of tumor cells, which ultimately manifest as reduced tumor proliferation and invasion, reduced lesion formation, and suppressed non-anchored growth [42]. This evidence concerns the gene AGTR1 and neoplasm.