AKT1 and infection: Specifically, enhanced pathways, including cytoskeleton proteins, focal adhesion, tight junction, proteoglycans, calcium signaling, tryptophan metabolism, tyrosine metabolism, ECM–receptor interaction, PI3K-Akt, Rap1, Ras, phagosome and pathogens infection-related, were shared in at least two groups out of NG, CG and SG (Figure 4 and Table S5).